Drug Monograph

Trulance (Plecanatide)

plecanatide

Guanylate Cyclase-C (GC-C) Agonist · Oral
Pharmacokinetic Profile
Half-Life
Not measurable (locally acting)
Metabolism
GI tract (loss of terminal leucine, then proteolysis)
Protein Binding
Negligible binding to albumin or α-1-acid glycoprotein
Bioavailability
Negligible systemic availability
Volume of Distribution
Not applicable (minimal tissue distribution)
Clinical Information
Drug Class
GC-C Agonist (Uroguanylin Analog)
Available Doses
3 mg tablet
Route
Oral
Renal Adjustment
Not required
Hepatic Adjustment
Not required
Pregnancy
Limited data; negligible absorption
Lactation
Not detected in breast milk (at 3 mg dose)
Schedule
Prescription only (not scheduled)
Generic Available
No (authorized generic available)
Black Box Warning
Yes — Pediatric dehydration risk (<6 years)
Rx

Indications

IndicationApproved PopulationTherapy TypeStatus
Chronic idiopathic constipation (CIC)Adults (≥18 years)MonotherapyFDA Approved
Irritable bowel syndrome with constipation (IBS-C)Adults (≥18 years)MonotherapyFDA Approved

Plecanatide was first approved by the FDA in January 2017 for CIC in adults, followed by the IBS-C indication in January 2018. It is the only GC-C agonist that is a structural analog of the endogenous hormone uroguanylin, and unlike linaclotide, it exhibits pH-sensitive receptor binding that preferentially activates GC-C in the mildly acidic environment of the proximal small intestine. The AGA issued a conditional recommendation for plecanatide in IBS-C based on moderate-certainty evidence (AGA 2022), and the AGA/ACG jointly recommended plecanatide strongly for CIC in adults who do not respond to over-the-counter agents (AGA/ACG 2023). Plecanatide does not have any approved pediatric indications.

Off-Label Uses

No well-established off-label indications exist for plecanatide. Anecdotal use parallels linaclotide off-label uses (e.g., opioid-induced constipation refractory to PAMORAs), but evidence is extremely limited. Evidence quality: Very low.

Dose

Dosing

Clinical ScenarioStarting DoseMaintenance DoseMaximum DoseNotes
CIC — adult3 mg once daily3 mg once daily3 mg/dayNo titration required; take with or without food, any time of day
6 mg dose did not add clinical benefit and increased adverse events in trials
IBS-C — adult3 mg once daily3 mg once daily3 mg/daySame dose as CIC; single strength simplifies prescribing
Addresses both abdominal pain and stool frequency in IBS-C
Swallowing difficulty — crushed tablet3 mg once daily3 mg once daily3 mg/dayCrush tablet and mix with 1 tsp applesauce or dissolve in 30 mL water; consume immediately
Can also be given via NG or gastric tube with 30 mL water
Clinical Pearl: Food Independence

A key practical advantage of plecanatide over linaclotide is that it can be taken with or without food, at any time of day. Linaclotide must be taken on an empty stomach at least 30 minutes before the first meal. For patients who find fasting requirements burdensome or who eat at irregular times, plecanatide offers greater dosing flexibility. The single 3 mg dose for both indications also eliminates the need to distinguish between CIC and IBS-C dosing.

PK

Pharmacology

Mechanism of Action

Plecanatide is a 16-amino-acid synthetic peptide that is structurally nearly identical to human uroguanylin, differing by only a single amino acid substitution designed to enhance GC-C binding affinity. Like its endogenous counterpart, plecanatide functions as a guanylate cyclase-C agonist, binding to GC-C receptors on the luminal surface of the intestinal epithelium. This binding is pH-sensitive, with higher affinity in the mildly acidic environment of the proximal small intestine (pH ~5–6), which mirrors how endogenous uroguanylin normally regulates intestinal fluid balance. Activation of GC-C increases both intracellular and extracellular cyclic guanosine monophosphate (cGMP). Intracellular cGMP stimulates chloride and bicarbonate secretion through the CFTR ion channel, increasing intestinal fluid and accelerating transit. Extracellular cGMP reduces the activity of visceral pain-sensing afferent neurons in animal models, contributing to abdominal pain relief in IBS-C.

ADME Profile

ParameterValueClinical Implication
AbsorptionNegligible systemic absorption; plasma concentrations below limit of quantitation in the majority of samples after 3 mg dosingStandard PK parameters (AUC, Cmax, t½) cannot be calculated; drug action is entirely local in the GI lumen
DistributionMinimally distributed to tissues; negligible binding to human serum albumin and α-1-acid glycoproteinNo dose adjustment required for renal or hepatic impairment; systemic off-target effects not expected
MetabolismMetabolized in the GI tract by loss of the terminal leucine to form an active metabolite; both parent and metabolite are proteolytically degraded to smaller peptides and amino acidsNo CYP2C9 or CYP3A4 inhibition; no CYP3A4 induction; not a P-gp or BCRP substrate or inhibitor
EliminationNo human excretion studies conducted; drug and metabolite not measurable in plasma at recommended dosesEntirely degraded within the intestinal lumen; renal and hepatic clearance pathways not relevant
SE

Side Effects

1–10% Common
Adverse EffectIncidenceClinical Note
Diarrhea5% (CIC); 4.3% (IBS-C)The only adverse effect reported at ≥2% in any trial; placebo rate was 1% in both populations; notably lower diarrhea rate than linaclotide (16–20%)
Nausea1–2% (IBS-C)Reported in 1–<2% of IBS-C patients at a rate exceeding placebo
Nasopharyngitis1–2% (IBS-C)Likely coincidental; observed in IBS-C trials
Upper respiratory tract infection<2% (both CIC and IBS-C)Reported at incidence exceeding placebo in both trial populations
Abdominal distension<2% (CIC)Noted in CIC trials at a rate above placebo
Flatulence<2% (CIC)Reported in CIC trials; self-limiting in most patients
Dizziness1–2% (IBS-C)Reported in the IBS-C trials at incidence exceeding placebo
Urinary tract infection1–2% (IBS-C)Likely coincidental; no plausible pharmacological mechanism
Serious Serious (Regardless of Frequency)
Adverse EffectEstimated FrequencyTypical OnsetRequired Action
Severe diarrhea with dehydration0.6% (CIC); 1% (IBS-C)Within first 3 days (CIC); within first day (IBS-C)Suspend dosing immediately; rehydrate orally or parenterally; monitor electrolytes
Elevated hepatic transaminases (ALT >5× ULN)Rare (2–3 patients per trial population)VariableMonitor LFTs; discontinue if clinically significant elevation persists; evaluate alternative causes
Hypersensitivity reactions (pruritus, urticaria, rash)Very rare (postmarketing)Any timeDiscontinue; standard allergy management; do not rechallenge if severe
Discontinuation Discontinuation Rates
CIC (Adults, 3 mg)
4% vs 2% placebo
Top reason: Diarrhea (2% vs 0.5% placebo)
IBS-C (Adults, 3 mg)
2.5% vs 0.4% placebo
Top reason: Diarrhea (1.2% vs 0% placebo)
Reason for DiscontinuationIncidenceContext
Diarrhea2% (CIC); 1.2% (IBS-C)Placebo comparator: 0.5% (CIC), 0% (IBS-C); notably lower discontinuation rate than linaclotide
Managing Diarrhea

Diarrhea is the primary tolerability concern with plecanatide, though its overall incidence (4–5%) and discontinuation rate (1–2%) are substantially lower than those reported with linaclotide (16–20% incidence, 2–5% discontinuation). Most cases begin within the first 4 weeks. In the CIC trials, severe diarrhea occurred within the first 3 days. If severe diarrhea occurs, suspend dosing and ensure adequate rehydration. Post-marketing experience has confirmed a low real-world diarrhea rate, with reports indicating a rate under 0.5% and no cases of severe diarrhea requiring hospitalization.

Int

Drug Interactions

Plecanatide has an exceptionally clean drug interaction profile due to its local GI activity and negligible systemic absorption. In vitro studies confirmed that neither plecanatide nor its active metabolite inhibits CYP2C9 or CYP3A4, induces CYP3A4, or acts as a substrate or inhibitor of P-glycoprotein or BCRP transporters. No formal drug-drug interaction studies were deemed necessary by the FDA. Additionally, a pooled analysis of Phase 3 CIC trial data demonstrated that concomitant use of acid suppression therapy (PPIs and H2RAs) did not diminish plecanatide efficacy or alter its safety profile.

ModerateOther secretagogues (linaclotide, lubiprostone)
MechanismAdditive intestinal chloride/fluid secretion via overlapping downstream pathways
EffectIncreased risk of diarrhea and potential electrolyte derangement
ManagementDo not combine GC-C agonists; switch between agents rather than stacking if one is ineffective
Clinical Rationale
ModerateAntidiarrheal agents (loperamide)
MechanismPharmacodynamic antagonism opposing secretory and prokinetic effect
EffectMay reduce plecanatide efficacy; concurrent use undermines therapeutic intent
ManagementAvoid routine coadministration; reassess indication rather than adding antidiarrheals
Clinical Rationale
MinorProton pump inhibitors / H2RAs
MechanismPlecanatide is pH-sensitive (higher GC-C affinity at lower pH); acid suppression raises intestinal pH
EffectTheoretical concern for reduced efficacy; pooled analysis showed no clinically significant impact
ManagementNo adjustment needed; safe to coadminister based on pooled Phase 3 data
Moshiree 2022
MinorOpioid analgesics
MechanismOpioids slow GI transit; pharmacodynamic opposition to prokinetic action
EffectReduced efficacy; no pharmacokinetic interaction
ManagementPlecanatide is not approved for OIC; PAMORAs remain the appropriate first-line for opioid-related constipation
Clinical Rationale
Key Interaction Profile Advantage

The absence of CYP, transporter, and systemic pharmacokinetic interactions, combined with confirmed safety alongside acid suppression therapy, makes plecanatide particularly suitable for patients on polypharmacy regimens. No dose adjustments are needed for any concomitant medication.

Mon

Monitoring

  • Stool Pattern Weeks 1–4, then as needed
    Routine
    Assess stool frequency, consistency, and presence of diarrhea. Most diarrhea begins within the first 4 weeks. In CIC trials, severe diarrhea occurred within the first 3 days. If severe, suspend dosing and rehydrate.
  • Hydration Status First 4 weeks, then as needed
    Routine
    Monitor for signs of dehydration, particularly in elderly patients and those on diuretics. Parenteral rehydration may be necessary in severe cases.
  • Hepatic Function If symptoms arise
    Trigger-based
    Elevated ALT (>5–15× ULN) and AST (>5× ULN) were reported in a small number of patients in clinical trials. Check LFTs if hepatic symptoms develop; no routine baseline LFTs are mandated by the label.
  • Abdominal Symptoms Baseline, then 4–8 weeks
    Routine
    Track abdominal pain severity and bloating in IBS-C patients. In trials, improvement in CSBM frequency was observed as early as week 1. Reassess treatment if no benefit after 4–6 weeks.
  • Treatment Efficacy 4–8 weeks after initiation
    Routine
    Evaluate whether the patient has met treatment goals for bowel frequency (target ≥3 CSBMs/week). In clinical trials, TRULANCE-treated patients returned to baseline within 2 weeks of stopping treatment.
CI

Contraindications & Cautions

Absolute Contraindications

  • Age <6 years: Contraindicated due to risk of fatal dehydration from GC-C agonism-induced fluid secretion in immature intestine (FDA Boxed Warning).
  • Known or suspected mechanical gastrointestinal obstruction: Secretory effects may worsen obstruction-related complications.

Relative Contraindications (Specialist Input Recommended)

  • Age 6 to <18 years: The FDA label explicitly states to avoid use in this population. Safety and efficacy have not been established in any pediatric age group. Although no deaths occurred in older juvenile mice, the lack of clinical data in children warrants avoidance.
  • Known hypersensitivity to plecanatide or any excipient: Pruritus, urticaria, and rash have been reported postmarketing.

Use with Caution

  • Elderly patients on diuretics or with limited fluid intake: Higher susceptibility to dehydration from drug-induced diarrhea.
  • Patients with inflammatory bowel disease: Not studied in IBD; theoretical concern that increased secretion could exacerbate diarrhea in active disease.
  • Pregnancy: No adequate human data, though negligible systemic absorption suggests low fetal risk. Use only if benefit justifies potential risk.
  • Lactation: Plecanatide and its metabolite were not detected in breast milk after 3 mg daily for 2 weeks, but potential for adverse effects in breastfed infants exists due to GC-C sensitivity in neonates.
FDA Boxed Warning Risk of Serious Dehydration in Pediatric Patients

Plecanatide is contraindicated in patients less than 6 years of age. In nonclinical studies, a single oral dose of plecanatide caused deaths due to dehydration in young juvenile mice (human age equivalent of approximately 1 month to less than 2 years) as a consequence of GC-C stimulation. Due to increased intestinal expression of GC-C, children under 6 years are at disproportionate risk of severe diarrhea and life-threatening dehydration. Use should also be avoided in patients 6 to less than 18 years of age given the absence of pediatric clinical data. Accidental ingestion by young children can be life-threatening; patients must store the medication securely out of reach of children.

Pt

Patient Counselling

Purpose of Therapy

Plecanatide works directly in the intestine to increase fluid secretion, soften stools, and speed up bowel transit. In patients with IBS-C, it also helps reduce abdominal pain by mimicking a natural hormone (uroguanylin) that regulates gut fluid. Most patients begin to notice improvement in stool frequency within the first week of treatment.

How to Take

Take one 3 mg tablet once daily, with or without food, at any convenient time of day. Swallow the tablet whole. If you cannot swallow tablets, you can crush the tablet and mix it with a teaspoon of applesauce (swallow immediately) or dissolve it in 30 mL of room-temperature water. If you miss a dose, skip it and take the next one at your regular time.

Diarrhea
Tell patientLoose stools can occur, most commonly within the first few weeks. This happens in about 1 in 20 patients and is usually mild. Staying well hydrated is important.
Call prescriberIf diarrhea becomes severe (very watery, frequent, or causing dizziness or faintness), stop the medication and contact your healthcare provider immediately.
Child Safety
Tell patientThis medication can cause severe, life-threatening dehydration in children under 6 years old. Keep the blister pack or bottle stored securely and completely out of reach of all children. Dispose of unused tablets safely.
Call prescriberIf a child accidentally swallows plecanatide, especially a child under 6 years, seek emergency medical care immediately.
Storage
Tell patientStore at room temperature in the original bottle or blister pack. Keep in a dry place and protect from moisture. For bottles, do not remove the desiccant packet. Remove and discard the polyester coil after first opening the bottle.
Call prescriberIf tablets appear damaged, discolored, or have been exposed to excessive moisture, consult your pharmacist.
Treatment Expectations
Tell patientPlecanatide works while you take it. If you stop, bowel symptoms typically return to baseline within about 2 weeks. This does not mean the condition has worsened — it simply means the treatment effect has ended. There is no rebound worsening beyond your original baseline.
Call prescriberIf you are considering stopping treatment, discuss alternatives or next steps with your provider first.
Ref

Sources

Regulatory (PI / SmPC)
  1. TRULANCE (plecanatide) tablets, for oral use. Full Prescribing Information. Salix Pharmaceuticals (originally Synergy Pharmaceuticals Inc.). Initial approval January 2017; IBS-C indication added January 2018; label updated through April 2021. DailyMedPrimary source for all dosing, adverse reaction rates, contraindications, and pharmacokinetic data. Lactation and postmarketing data from 2020–2021 label revisions.
  2. Trulance (plecanatide) FDA Approval History. Drugs.com. Accessed April 2026. Drugs.comTimeline of FDA regulatory actions from CIC approval (Jan 2017) through IBS-C approval (Jan 2018).
Key Clinical Trials
  1. Miner PB Jr, Koltun WD, Wiener GJ, et al. A randomized phase III clinical trial of plecanatide, a uroguanylin analog, in patients with chronic idiopathic constipation. Am J Gastroenterol. 2017;112(4):613–621. DOIPivotal Phase 3 CIC trial (Study 1, N=1394) establishing efficacy of plecanatide 3 mg (21% vs 10.2% placebo responder rate).
  2. DeMicco M, Barrow L, Hickey B, et al. Randomized clinical trial: efficacy and safety of plecanatide in the treatment of chronic idiopathic constipation. Therap Adv Gastroenterol. 2017;10(11):837–851. DOISecond pivotal Phase 3 CIC trial (Study 2, N=1337) confirming durable CSBM response (21% vs 13% placebo).
  3. Brenner DM, Fogel R, Dorn SD, et al. Efficacy, safety, and tolerability of plecanatide in patients with irritable bowel syndrome with constipation: results of two phase 3 randomized clinical trials. Am J Gastroenterol. 2018;113(5):735–745. DOITwo pivotal Phase 3 IBS-C trials (Studies 3 and 4, N=2189 total) establishing combined responder rates of 30% and 21% vs placebo.
  4. Brenner DM, Sharma A, Rao SSC, et al. Plecanatide improves abdominal bloating and bowel symptoms of irritable bowel syndrome with constipation. Dig Dis Sci. 2024;69(5):1731–1738. DOIPooled Phase 3 post-hoc analysis demonstrating plecanatide efficacy in IBS-C patients with moderate-to-severe bloating.
Guidelines
  1. Chang L, Sultan S, Lembo A, et al. AGA Clinical Practice Guideline on the pharmacological management of irritable bowel syndrome with constipation. Gastroenterology. 2022;163(1):118–136. DOIConditional recommendation for plecanatide in IBS-C based on moderate-certainty evidence.
  2. Chang L, Chey WD, Imdad A, et al. AGA–ACG Clinical Practice Guideline: pharmacological management of chronic idiopathic constipation. Gastroenterology. 2023;164(7):1086–1106. DOIStrong recommendation for plecanatide for CIC after OTC agent failure, based on moderate-certainty evidence.
  3. Lacy BE, Pimentel M, Brenner DM, et al. ACG Clinical Guideline: management of irritable bowel syndrome. Am J Gastroenterol. 2021;116(1):17–44. DOIACG guideline recommending GC-C agonists (including plecanatide) for IBS-C based on high-quality evidence.
Mechanistic / Basic Science
  1. Shailubhai K, Comiskey S, Foss JA, et al. Plecanatide, an oral guanylate cyclase C agonist acting locally in the gastrointestinal tract, is safe and well-tolerated in single doses. Dig Dis Sci. 2013;58(9):2580–2586. DOIPhase 1 study confirming local GI action, negligible systemic absorption, and safety in healthy volunteers.
  2. Boulete IM, Thadi A, Beaufrand C, et al. Oral treatment with plecanatide or dolcanatide attenuates visceral hypersensitivity via activation of guanylate cyclase-C in rat models. World J Gastroenterol. 2018;24(17):1888–1900. DOIPreclinical evidence that GC-C agonism with plecanatide reduces visceral pain signaling in animal models.
Pharmacokinetics / Special Populations
  1. Moshiree B, Schoenfeld P, Franklin H, et al. The effect of acid suppression therapy on the safety and efficacy of plecanatide: analysis of randomized phase III trials. Clin Ther. 2022;44(1):98–110.e1. DOIPooled Phase 3 analysis confirming that concomitant PPI/H2RA use does not affect plecanatide efficacy or safety.
  2. Menees SB, Franklin H, Chey WD. Evaluation of plecanatide for the treatment of chronic idiopathic constipation and irritable bowel syndrome with constipation in patients 65 years or older. Clin Ther. 2020;42(7):1406–1414.e4. DOIPooled Phase 3 analysis in elderly patients (≥65 years) confirming comparable efficacy and safety to the younger population.