Drug Monograph

Ampicillin-Sulbactam (Unasyn)

ampicillin sodium / sulbactam sodium

Aminopenicillin / Beta-Lactamase Inhibitor · Intravenous · Intramuscular
Pharmacokinetic Profile
Half-Life
~1 h (both agents)
Metabolism
Minimal hepatic metabolism
Protein Binding
Amp 28%; Sulb 38%
Bioavailability
IV: 100%; IM: well absorbed
Elimination
Renal: 75–85% unchanged in urine
Clinical Information
Drug Class
Aminopenicillin / BLI combination
Available Doses
1.5 g (1 g + 0.5 g), 3 g (2 g + 1 g) vials
Route
IV and IM
Renal Adjustment
Required (CrCl <30 mL/min)
Hepatic Adjustment
Not required; monitor LFTs
Pregnancy
No evidence of harm in animal studies; use if clearly needed
Lactation
Low concentrations in breast milk; use with caution
Schedule
Prescription only (not scheduled)
Generic Available
Yes
Sodium Content
115 mg (5 mEq) Na+ per 1.5 g
Rx

Indications

IndicationApproved PopulationTherapy TypeStatus
Skin and skin structure infections (cellulitis, cutaneous abscesses, wound infections)Adults and pediatric ≥1 yearMonotherapyFDA Approved
Intra-abdominal infections (peritonitis, intra-abdominal abscess)AdultsMonotherapyFDA Approved
Gynecological infections (endometritis, PID)AdultsMonotherapyFDA Approved

Ampicillin-sulbactam combines an aminopenicillin with a beta-lactamase inhibitor, restoring activity against many beta-lactamase-producing organisms including S. aureus (MSSA), E. coli, Klebsiella spp., H. influenzae, Bacteroides fragilis, and Acinetobacter species. It retains ampicillin’s inherent activity against enterococci, streptococci, and Listeria. It does not cover MRSA, Pseudomonas aeruginosa, or ESBL-producing Enterobacterales.

Off-Label Uses

Community-acquired aspiration pneumonia: Frequently used for polymicrobial aspiration pneumonia due to excellent anaerobic coverage. Evidence quality: Moderate.

Human and animal bite wounds: Recommended by IDSA guidelines for bite-wound infections covering oral flora including Pasteurella, Eikenella, and anaerobes. Evidence quality: High.

Deep neck space infections (Ludwig’s angina, peritonsillar abscess): Commonly used as empiric therapy for polymicrobial head and neck infections. Evidence quality: Moderate.

Carbapenem-resistant Acinetobacter baumannii (CRAB): IDSA 2022 guidance recommends high-dose ampicillin-sulbactam (9 g IV q8h over 4-hour infusion) as sulbactam has intrinsic activity against Acinetobacter via PBP binding. Evidence quality: Moderate.

Diabetic foot infections (mild-moderate): Covers polymicrobial flora including MSSA, streptococci, and anaerobes. Evidence quality: Moderate.

Dose

Dosing

Adult Dosing by Clinical Scenario

Clinical ScenarioStarting DoseMaintenance DoseMaximum DoseNotes
Skin / soft tissue infection (mild–moderate)1.5–3 g IV/IM q6h1.5–3 g IV/IM q6h12 g/day (sulbactam max 4 g/day)Duration 7–14 days
IV over 10–15 min (bolus) or 15–30 min (infusion); IM via deep injection
Intra-abdominal infection (complicated appendicitis, peritonitis)3 g IV q6h3 g IV q6h12 g/dayDuration 5–14 days depending on source control
Use higher dose (3 g) for serious infections
Gynecological infection (endometritis, PID)3 g IV q6h3 g IV q6h12 g/dayDuration guided by clinical response
Consider adding doxycycline for PID per CDC guidelines
Bite wound infection (human/animal) — off-label1.5–3 g IV q6h1.5–3 g IV q6h12 g/dayStep down to oral amoxicillin-clavulanate when clinically appropriate
Covers Pasteurella, Eikenella, oral anaerobes
Aspiration pneumonia — off-label3 g IV q6h3 g IV q6h12 g/dayDuration 5–7 days typically
Excellent anaerobic coverage for aspiration
CRAB infection — high-dose sulbactam (off-label)9 g IV q8h9 g IV q8h27 g/dayInfuse over 4 hours; exceeds standard sulbactam cap
Per IDSA 2022 CRAB guidance; sulbactam provides PBP-mediated activity against A. baumannii

Renal Impairment Dosing — Adults (FDA PI)

CrCl (mL/min)Starting DoseMaintenance DoseMaximum DoseNotes
≥301.5–3 g q6–8h1.5–3 g q6–8h12 g/dayNo dose adjustment required
15–291.5–3 g q12h1.5–3 g q12h6 g/dayExtend interval
5–141.5–3 g q24h1.5–3 g q24h3 g/dayExtend interval further
Hemodialysis1.5–3 g q24h1.5–3 g q24h3 g/dayAdminister after dialysis on dialysis days
Both agents removed by hemodialysis

Pediatric Dosing (≥1 year, <40 kg)

Clinical ScenarioStarting DoseMaintenance DoseMaximum DoseNotes
Skin and skin structure infections (FDA-approved)300 mg/kg/day IV divided q6h300 mg/kg/day IV divided q6hAdult dose if ≥40 kg= 200 mg/kg ampicillin + 100 mg/kg sulbactam per day
IV only for pediatrics; max treatment 14 days IV, then switch to oral
Clinical Pearl: Sulbactam Maximum and CRAB Dosing

The FDA-approved maximum sulbactam dose is 4 g/day (corresponding to 12 g/day total Unasyn). However, for CRAB infections, the IDSA 2022 guidance recommends doses far exceeding this limit — 9 g total (6 g ampicillin + 3 g sulbactam) every 8 hours, infused over 4 hours. This high-dose strategy leverages sulbactam’s intrinsic bactericidal activity against A. baumannii via binding to PBP1 and PBP3, independent of beta-lactamase inhibition. This is a fundamentally different pharmacological rationale from standard use.

PK

Pharmacology

Mechanism of Action

Ampicillin is an aminopenicillin that exerts bactericidal activity by binding to penicillin-binding proteins (PBPs) in the bacterial cell wall, inhibiting the final transpeptidation step of peptidoglycan synthesis. This leads to cell wall weakening, osmotic instability, and bacterial lysis during active growth. Ampicillin has broad activity against gram-positive organisms (streptococci, enterococci, Listeria), many gram-negative aerobes (E. coli, Proteus mirabilis, H. influenzae), and anaerobes. However, ampicillin is readily hydrolysed by beta-lactamases, limiting its utility against resistant organisms. Sulbactam is a penicillanic acid sulfone that irreversibly inhibits many plasmid-mediated and chromosomal class A beta-lactamases, preventing them from inactivating ampicillin. While sulbactam has minimal intrinsic antibacterial activity against most organisms, it does possess clinically meaningful direct activity against Neisseria species and, importantly, against Acinetobacter baumannii through direct PBP binding — a property exploited in high-dose CRAB protocols.

ADME Profile

ParameterValueClinical Implication
AbsorptionIV: peak levels immediately post-infusion (amp 109–150 mcg/mL at 2 g dose); IM: peak amp 8–37 mcg/mLIM route available for outpatient settings; IV preferred for serious infections
DistributionAmp 28% protein bound; sulb 38%; good penetration to peritoneal fluid, intestinal mucosa, appendix, blister fluid; CSF penetration with inflamed meningesTissue levels generally adequate for intra-abdominal and soft tissue infections; can reach CSF in meningitis
MetabolismMinimal; both agents undergo negligible hepatic metabolismNo hepatic dose adjustment; however, monitor LFTs given hepatotoxicity risk
Eliminationt½ ~1 h (both agents); 75–85% excreted unchanged in urine within 8 h; both removable by hemodialysisRenal dose adjustment required when CrCl <30 mL/min; dose after dialysis
SE

Side Effects

≥10%Very Common
Adverse EffectIncidenceClinical Note
Pain at IM injection site16%Expected with IM administration; can be reduced by using lidocaine as diluent where permitted
1–10%Common
Adverse EffectIncidenceClinical Note
Diarrhea3%Most common systemic adverse reaction; usually mild; evaluate for C. difficile if severe or bloody
Pain at IV injection site3%Can be minimized by slower infusion and adequate dilution
Thrombophlebitis3%Rotate IV sites; consider midline or PICC for prolonged courses
Rash<2%Distinguish allergic rash from non-allergic ampicillin rash (5–10% in children, especially with mononucleosis); non-allergic rash is dull red, maculopapular, appears day 3–14
Phlebitis1.2%Related to infusion; use adequate dilution volume
SeriousSerious (Regardless of Frequency)
Adverse EffectEstimated FrequencyTypical OnsetRequired Action
AnaphylaxisRareMinutes to first hourStop immediately; epinephrine; airway management; permanent discontinuation
Stevens-Johnson syndrome / TENVery rare1–4 weeksDiscontinue immediately; dermatology consultation; supportive care
DRESSVery rare2–8 weeksDiscontinue; systemic corticosteroids; monitor end-organ function
Hepatotoxicity (cholestatic jaundice, hepatitis)Uncommon; deaths reportedDays to weeksMonitor LFTs; discontinue if jaundice or significant transaminase elevation; contraindicated on re-exposure
Clostridioides difficile–associated diarrheaUncommonDuring therapy to ≥2 months afterTest for toxin; discontinue ampicillin-sulbactam if possible; treat with vancomycin or fidaxomicin per guidelines
Hemolytic anemiaVery rare (postmarketing)VariableDiscontinue; supportive care; usually reversible
Agranulocytosis / thrombocytopenic purpuraVery rare (postmarketing)Typically with prolonged therapyDiscontinue; monitor CBC; usually reversible
SeizuresVery rare; higher risk with renal impairmentVariable; dose-relatedEnsure appropriate renal dosing; benzodiazepines for acute seizure
Acute generalized exanthematous pustulosis (AGEP)Very rare (postmarketing)Days to weeksDiscontinue; dermatology consultation; usually self-resolving after cessation
DiscontinuationDiscontinuation Rates
Clinical Trials (Adults)
Low overall discontinuation rate
Context: The FDA PI describes Unasyn as “generally well tolerated”; specific overall discontinuation rates are not separately quantified in the label, but adverse events leading to cessation were infrequent in clinical trials.
Pediatric Trials
Similar safety profile to adults
Context: Available data show comparable adverse event rates in children ≥1 year. Non-allergic ampicillin rash occurs in 5–10% of children.
Reason for DiscontinuationIncidenceContext
Rash / dermatologic reactions<2%Evaluate for allergic vs non-allergic ampicillin rash
GI intolerance (diarrhea, nausea)~3% affected; rarely requires cessationUsually mild and self-limiting
HepatotoxicityRareWarrants permanent discontinuation; re-exposure is contraindicated
Managing Ampicillin Rash in Children

A dull, red maculopapular rash occurs in 5–10% of children receiving ampicillin-class drugs, typically appearing on day 3–14 of therapy. This non-allergic rash is particularly common in patients with infectious mononucleosis (up to 70–100% incidence), in whom ampicillin-class agents should be avoided. The rash is not a true allergy and does not preclude future penicillin use, but careful clinical evaluation is needed to distinguish it from urticarial or anaphylactic hypersensitivity.

Int

Drug Interactions

Ampicillin-sulbactam undergoes minimal hepatic metabolism and is not a significant CYP450 inducer or inhibitor. Its key interactions relate to renal excretion competition, physical incompatibility with aminoglycosides, and pharmacodynamic effects on coagulation and gut flora.

MajorMethotrexate
MechanismPenicillins reduce renal tubular secretion of methotrexate
EffectElevated methotrexate levels with increased haematologic and GI toxicity
ManagementMonitor methotrexate levels closely; consider leucovorin rescue; avoid combination if possible
Lexicomp
ModerateProbenecid
MechanismInhibits renal tubular secretion of both ampicillin and sulbactam
EffectProlonged half-life and higher serum levels
ManagementMonitor for toxicity; may be used intentionally to prolong levels in specific situations
FDA PI
ModerateAllopurinol
MechanismUnknown; possibly hyperuricemia-related
EffectSubstantially increased incidence of rash with concurrent ampicillin
ManagementMonitor for rash; consider alternative antibiotic if rash develops
FDA PI
ModerateAminoglycosides
MechanismIn vitro inactivation of aminoglycosides by ampicillin
EffectReduced aminoglycoside efficacy if mixed in the same solution
ManagementDo not reconstitute or mix together; administer via separate IV lines or sequentially with line flush
FDA PI
ModerateOral Contraceptives
MechanismDisruption of enterohepatic recirculation of oestrogens by alteration of gut flora
EffectPotentially reduced contraceptive efficacy
ManagementAdvise backup contraception during therapy and for 7 days after completion
Lexicomp
MinorWarfarin / Anticoagulants
MechanismBroad-spectrum antibiotics may alter vitamin K-producing gut flora
EffectPotentially enhanced anticoagulant effect; elevated INR
ManagementMonitor INR more frequently during therapy
Lexicomp
MinorLaboratory Test Interference
MechanismNon-enzymatic urine glucose methods
EffectFalse-positive urine glucose (Clinitest); false decrease in plasma conjugated estriol in pregnancy
ManagementUse enzymatic glucose methods; be aware of estriol assay interference in pregnancy monitoring
FDA PI
Mon

Monitoring

  • Hepatic FunctionBaseline; periodically during therapy
    Routine
    AST, ALT, alkaline phosphatase, bilirubin. Hepatotoxicity including cholestatic jaundice has been reported with deaths; monitor at regular intervals, especially in patients with pre-existing hepatic impairment
  • Renal FunctionBaseline; then as indicated
    Routine
    BUN, creatinine; adjust dosing interval when CrCl <30 mL/min; increases in BUN and creatinine reported in clinical trials
  • CBCBaseline; weekly if >7 days
    Routine
    Monitor for leukopenia, neutropenia, thrombocytopenia, eosinophilia; agranulocytosis and hemolytic anemia reported in postmarketing
  • Signs of Allergy / SkinEach dose; ongoing
    Routine
    First dose: monitor for anaphylaxis. Ongoing: assess for progressive rash, mucosal involvement (SJS/TEN), or systemic symptoms (DRESS). Discontinue for progressive lesions
  • GI SymptomsOngoing; test if severe diarrhea
    Trigger-based
    Test for C. difficile toxin if diarrhea is watery, bloody, or persistent; can occur up to 2+ months post-treatment
  • SuperinfectionOngoing
    Trigger-based
    Monitor for oral or vaginal candidiasis and resistant bacterial superinfection during prolonged courses
CI

Contraindications & Cautions

Absolute Contraindications

  • History of serious hypersensitivity (anaphylaxis, SJS) to ampicillin, sulbactam, any penicillin, or other beta-lactam antibacterials (including cephalosporins)
  • Previous cholestatic jaundice or hepatic dysfunction associated with ampicillin-sulbactam use — re-exposure is specifically contraindicated per FDA PI

Relative Contraindications (Specialist Input Recommended)

  • Infectious mononucleosis: A high percentage of patients with mononucleosis develop a generalized maculopapular rash when given ampicillin-class agents; avoid use in known or suspected mono
  • Known cephalosporin allergy (severe): Cross-reactivity estimated at 1–2%; specialist assessment recommended for severe previous cephalosporin reactions

Use with Caution

  • Renal impairment (CrCl <30 mL/min): Dose interval extension required; both agents accumulate; increased risk of neurotoxicity (seizures) at elevated drug concentrations
  • Pre-existing hepatic disease: Monitor hepatic function at regular intervals; hepatotoxicity (cholestatic jaundice) has resulted in fatal outcomes
  • Concurrent allopurinol therapy: Substantially increased rash incidence
  • Patients on oral anticoagulants: Monitor coagulation more frequently
  • Sodium-restricted diets: Significant sodium load (5 mEq per 1.5 g; up to 40 mEq/day at full dose of 12 g/day)
FDA Safety Warning Hepatotoxicity

Hepatic dysfunction, including hepatitis and cholestatic jaundice, has been associated with ampicillin-sulbactam use. While usually reversible, fatal cases have been reported. Hepatic function should be monitored at regular intervals in patients with hepatic impairment. A previous episode of cholestatic jaundice or hepatic dysfunction attributable to ampicillin-sulbactam is a contraindication to re-exposure.

FDA Safety Warning Severe Cutaneous Adverse Reactions

Ampicillin-sulbactam has been associated with severe skin reactions including toxic epidermal necrolysis (TEN), Stevens-Johnson syndrome (SJS), drug reaction with eosinophilia and systemic symptoms (DRESS), dermatitis exfoliative, erythema multiforme, and acute generalized exanthematous pustulosis (AGEP). Any progressive skin rash should prompt immediate discontinuation and evaluation.

Pt

Patient Counselling

Purpose of Therapy

Ampicillin-sulbactam is an antibiotic given through a vein or by injection to treat serious bacterial infections. It works by killing bacteria and preventing them from resisting treatment. It is not effective against viral infections such as the common cold or influenza.

How to Take

This medication is administered by healthcare professionals, usually every 6 hours by IV infusion (over 15–30 minutes) or by deep injection into a muscle. Each scheduled dose is important — completing the full course is essential even if symptoms improve early, as stopping prematurely increases the risk of resistant infections.

Diarrhea
Tell patientLoose stools are common during antibiotic treatment and usually resolve after stopping. Stay hydrated and inform the care team if diarrhea is frequent.
Call prescriberIf stools become watery, bloody, or are accompanied by stomach cramps and fever — even weeks after the last dose. Do not take anti-diarrhea medicines without checking first.
Allergic Reactions & Rash
Tell patientMild rash can occur and is usually not dangerous. Inform the care team of any previous antibiotic allergies before the first dose. A dull red rash appearing several days into treatment may be a non-allergic ampicillin rash and does not necessarily indicate a true allergy.
Call prescriberImmediately alert staff if experiencing difficulty breathing, throat tightness, facial swelling, or widespread hives. Any blistering, peeling skin, or mouth sores require urgent evaluation.
Injection Site Discomfort
Tell patientPain, redness, or swelling at the injection site is common, especially with intramuscular injections. Inform nursing staff so they can rotate sites or adjust the infusion.
Call prescriberIf the area becomes increasingly swollen, warm, or red with streaking along the vein (possible phlebitis or thrombophlebitis).
Completing the Full Course
Tell patientFeeling better before the antibiotic course is finished is expected. Stopping early allows surviving bacteria to multiply and develop resistance, potentially making future infections harder to treat.
Call prescriberIf symptoms are not improving within 48–72 hours, or if new symptoms such as worsening pain, high fever, or confusion develop.
Ref

Sources

Regulatory (PI / SmPC)
  1. Unasyn (ampicillin sodium and sulbactam sodium) for Injection. Full Prescribing Information. Pfizer Inc. Revised May 2024. FDA Label (PDF) Primary source for all approved indications, dosing, renal adjustment, adverse reactions, contraindications, and drug interactions.
  2. Unasyn (ampicillin sodium and sulbactam sodium). DailyMed label. National Library of Medicine. DailyMed Current NLM-hosted label with full prescribing information and clinical pharmacology data.
  3. Unasyn (ampicillin sodium and sulbactam sodium) for Injection. Updated Prescribing Information January 2025. FDA Label 2025 (PDF) Most recent FDA label update with current warnings including severe cutaneous adverse reactions.
Key Clinical Trials
  1. Yellin AE, Heseltine PN, Berne TV, et al. The role of sulbactam/ampicillin in the treatment of serious surgical infections. Surg Gynecol Obstet. 1993;177(Suppl):30-40. Pivotal trial supporting efficacy of ampicillin-sulbactam in intra-abdominal and surgical infections.
  2. Betrosian AP, Frantzeskaki F, Xanthaki A, Georgiadis G. High-dose ampicillin-sulbactam as an alternative treatment of late-onset VAP from multidrug-resistant Acinetobacter baumannii. Scand J Infect Dis. 2007;39(1):38-43. doi:10.1080/00365540600951184 Evidence supporting high-dose sulbactam strategy for MDR Acinetobacter infections.
Guidelines
  1. Tamma PD, Aitken SL, Bonomo RA, Mathers AJ, van Duin D, Clancy CJ. IDSA 2023 guidance on the treatment of antimicrobial-resistant gram-negative infections. Clin Infect Dis. 2023. doi:10.1093/cid/ciad428 IDSA guidance recommending high-dose ampicillin-sulbactam (9 g q8h over 4 h) for CRAB infections.
  2. Stevens DL, Bisno AL, Chambers HF, et al. Practice guidelines for the diagnosis and management of skin and soft tissue infections: 2014 update by the IDSA. Clin Infect Dis. 2014;59(2):e10-52. doi:10.1093/cid/ciu296 IDSA guidelines listing ampicillin-sulbactam as an option for bite wounds and polymicrobial soft tissue infections.
  3. Solomkin JS, Mazuski JE, Bradley JS, et al. Diagnosis and management of complicated intra-abdominal infection in adults and children: guidelines by SIS and IDSA. Clin Infect Dis. 2010;50(2):133-164. doi:10.1086/649554 Recommends ampicillin-sulbactam as one option for community-acquired intra-abdominal infections of mild-moderate severity.
Mechanistic / Basic Science
  1. Bush K, Jacoby GA. Updated functional classification of beta-lactamases. Antimicrob Agents Chemother. 2010;54(3):969-976. doi:10.1128/AAC.01009-09 Comprehensive beta-lactamase classification relevant to sulbactam’s spectrum of inhibition.
  2. Penwell WF, Shapiro AB, Giacobbe RA, et al. Molecular mechanisms of sulbactam antibacterial activity and resistance determinants in Acinetobacter baumannii. Antimicrob Agents Chemother. 2015;59(3):1680-1689. doi:10.1128/AAC.04808-14 Elucidates sulbactam’s direct PBP-mediated bactericidal activity against A. baumannii, the basis for high-dose CRAB protocols.
Pharmacokinetics / Special Populations
  1. Peechakara BV, Gupta M. Ampicillin/Sulbactam. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2024 Jan. Updated January 11, 2024. NCBI Bookshelf Comprehensive review covering pharmacology, indications, adverse effects, and drug interactions with current references.
  2. Ito A, Kohno S, Nishizawa K, et al. Population pharmacokinetics of ampicillin and sulbactam in patients with community-acquired pneumonia: evaluation of the impact of renal impairment. Br J Clin Pharmacol. 2015;79(4):681-691. doi:10.1111/bcp.12533 Population PK model supporting renal dose adjustments and time-above-MIC analysis for various degrees of renal impairment.